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- A ketogenic diet, which calls for minimizing carbohydrates and replacing them with healthy fats and moderate amounts of high quality protein, can offer hope against cancer, both for prevention and treatment
- Your normal cells have the metabolic flexibility to adapt from using glucose to using ketone bodies. Cancer cells lack this ability so when you reduce carbs to only non-starchy vegetables, you effectively starve the cancer
- Cancer can be more accurately classified as a mitochondrial metabolic disease. Few people inherit genes that predispose them to cancer (most inherit genes that prevent cancer), and inherited mutations typically disrupt the function of the mitochondria
- The mitochondria—the main power generators in your cells—are believed to be the central point in the origins of many cancers. Your mitochondria can be damaged not only by inherited mutations, but also by a wide variety of environmental factors and toxins
- Fasting has remarkable health benefits and strengthens your mitochondria network systems throughout your body. As long as your mitochondria remain healthy and functional, it’s very unlikely that cancer will develop
A ketogenic diet calls for eliminating all but non-starchy vegetable carbohydrates, and replacing them with healthy fats and high quality protein.
The premise is that since cancer cells need glucose to thrive, and carbohydrates turn into glucose in your body, then lowering the glucose level in your blood though carb and protein restriction, literally starves the cancer cells into oblivion. Additionally, low protein intake tends to minimize the mTOR pathway that accelerates cell proliferation.
This type of diet, in which you restrict all but non-starchy vegetable carbs and replace them with low to moderate amounts of high quality protein and high amounts of beneficial fat, is what I recommend for everyone, whether you have cancer or not. It’s a diet that will help optimize your weight and all chronic degenerative disease. Eating this way will help you convert from carb burning mode to fat burning.
Dr. Thomas Seyfried is one of the leading pioneer academic researchers in promoting how to treat cancer nutritionally. He’s been teaching neurogenetics and neurochemistry as it relates to cancer treatment at Yale University and Boston College for the past 25 years.
He’s written over 150 peer-reviewed scientific articles and book chapters, and has also published a book, Cancer as a Metabolic Disease: On the Origin, Management, and Prevention of Cancer.
“We’re not going to make major advances in the management of cancer until it becomes recognized as a metabolic disease. But in order to do that, you have to present a massive counterargument against the gene theory of cancer,” he says.
“One of the key issues here is that if you transplant the nucleus of a cancer cell into a normal cell, you don’t get cancer cells. You can actually get normal tissues and sometimes a whole normal organism from the nucleus of a cancer cell. Now, if the tumors are being driven by driver genes – all these kinds of mutations and things that we hear about – how is it possible that all of this is changed when you place this cancer nucleus into the cytoplasm of a cell with normal mitochondria?
The gene theory cannot address this. It clearly argues strongly against the concept that genes are driving this process. Actually, a very few people inherit genes that predispose them to cancer. Most people inherit genes that prevent cancer. And those few genes that are inherited – the germ line like the BRCA1 mutations, B53, and a few other very rare cancers – these inherited mutations appear to disrupt the function of the mitochondria.”
“When we’re dealing with glucose and [cancer] management, we know from a large number of studies that if respiration of the tumor is ineffective, in order to survive, the cells must use an alternative source of energy, which is fermentation. We know that glucose is the primary fuel for fermentation. Fermentation becomes a primary energy-generating process in the tumor cell. By targeting the fuel for that process, we then have the capability of potentially managing the disease.”
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